This is a specific subject page, dealing exclusively with, or primarily with, the subject in the title. Because of need, there are many such pages at RHWW: usually, but not always, linked to primary pages. For those in a hurry, they enable a quick summary of many important subjects. The menu for these pages is here: Click>>>

Albino and Mulatto Mongols:

and their Sad, rather Pathetic, Race confusion

But first: It is obviously very nice to have total control of your spoken language, whether or not you created it, the world scientific community, and all methods of media and communication to disperse what you have devised in those disciplines. But regardless of those advantages/disadvantages for us; we here at RealHistoryww continue to try to keep up with the Albino peoples latest Race lies. Often, all we need do is simply dig down to find the true meaning of their latest "Made-up" lie word or term. At other times we simply have to find "Old" scientific studies that the new liars were not aware of, which contradict the new lies.

Gold is considered the most beautiful color to Chinese/Mongols:

things termed "Golden" are the greatest manifestation of those things,

in other words, the "Ultimate" of those things.

So consider this definition of White Skin...

So clearly the Chinese scientists responsible for this silly Albino adoring Study:

both of whom are from the "Chinese Academy of Sciences"

are saying that Albinism (Whiteness) is the "Height of Human Evolution"!

If that is true, then why would EVOLUTION leave White People with NO SAFE PLACE TO LIVE ON PLANET EARTH? Lets look at the EVIDENCE: Even the coldest, most Sun-Deprived place on the Earth "Antarctic" is not safe for White people as regards the Sun!

Antarctic researchers need solid sun block: study. Expeditioners to Antarctic train for freezing temperatures and social isolation, but a study has found there is something else to be wary of -- SUNBURN! The recent joint study by the Australian Antarctic Division (AAD) and the Australian Radiation Protection and Nuclear Safety Agency found that more than 80 percent of researchers to the South Pole were potentially exposed to ultraviolet (UV) rays in excess of the recommended limits. Almost a third received more than five times these limits.

The study showed that in some cases the UV exposure levels in Australian Antarctic stations can reach an index level of 8 or more, making exposure levels there similar to what lifeguards in Australia's sunny Queensland state potentially receive.

http://www.reuters.com/article/2009/12/21/us-antarctica-sunburn-idUSTRE5BK10120091221

Since ALL Humans (including Europeans) "Evolved" in Africa, just look at the result of light skin in Africa.

(These are the "Less" gruesome images of Skin Cancer)

Mother Nature would NOT do that!

Nature creates Advantageous Mutations, not Killer Mutations, only disease does that!

And that is exactly what "lack of Pigmentation" (Albinism) is - a disease.

_____________________________________________________________________________________________

For years Albino media hid the terrible truth from European Albinos,

they showed European Albinos out in the Sun, with not a care in the World, just as if they were Black people.

It is only now that European Albinos are starting to accept, and tell, the truth about their limitations in the Sun.

From GeekPrepper.org....

Sun burn is bad even during the best of times. Too much sun can lead to illness and sun stroke, and can be "FATAL". What are our options for sun protection? What if you are caught in the sun unexpectedly and need to be on the move? {A humorous coincidence, the motion picture was likely made well before they were born}.

Mud: You can smear mud over exposed skin for added sun protection and to prevent sunburn. Mud acts as a physical barrier sunscreen, because it prevents the sun’s UV rays from contacting or penetrating your skin. Mud will stick to your skin and then likely crack and fall off, leaving behind a dirty residue that can also block UV rays. If you find yourself without proper clothing or any other source of sunscreen, covering yourself with mud or dirt or any other similar opaque substance will help reflect the sun’s rays from your skin and prevent sunburn. Examples from the Discovery Channel’s Naked & Afraid program....

Synthetic “Melanin”

From Scientific American: Researchers at the University of California, San Diego, bathed dopamine—a signaling chemical found in the brain and other parts of the body—in an alkaline solution. This step produced melaninlike nanoparticles with shells and cores made of polydopamine, a dopamine-based polymer. When incubated in a petri dish with human keratinocytes, the synthetic particles were absorbed by the skin cells and distributed around their nuclei like natural melanin.

While we wait for this science to be perfected, so that we can all be Black, and Natural to Planet Earth again. The only current way back to Black is in the steps of "True Natural Selection". That is the process of finding a Black person to mate with, and produce "Partially Melaninated" offspring – which is better than having no or very little Melanin.

Note in the example below: though this "Partially Melaninated" Chinese young man "Did" suffer skin damage from the Sun, his skin did NOT "Blister" and become infected. Just the top layer of skin was killed, and thereby, was capable of being peeled away. (It would probably NOT be a good idea to do that often).

The Miracle of Melanin

The "Physiology" of WHY that young Mulatto Chinese mans sunburned skin did NOT Blister and become infected, is complicated: and science is still trying to understand all the benefits of having Melanin in your body. But one thing that we do know is that with Melanin, we also get a super advanced system of Skin Repair. You see, strange as it seems, Black People actually DO get Sunburned! But with Black people, only the uppermost layer of skin is killed, and it appears as a white "ashy" layer on the skin: which an application of skin cream instantly removes.

All of this happens because black skin (Melanin) enhances the body’s natural immune system and provides better protection against disease than white skin (Mackintosh, 2001). The melanin in dark skin is believed to help enhance the body’s natural ability to combat pathogens (Nosanchuk and Casadevall, 2006; Mackintosh, 2001; Manning et al, 2003). A major function of melanocytes, melanosomes and melanin in skin is to inhibit the proliferation of bacterial, fungal and other parasitic infections of the dermis and epidermis (Mackintosh, 2001).

The melanization of skin and other tissues form an important component of the innate immune defense system (Mackintosh, 2001; Nosanchuk and Casadevall, 2006). Protection from bacterial infection is also believed to be the reason that melanocyte and melanization patterns among different parts of the body do not reflect exposure to sunlight (Mackintosh, 2001). For example, many parts of the body which are hardly ever exposed to sunlight, such as genitalia, throats and nasal passages are often packed with melanin cells. In addition, several types of cells that are unrelated to skin also accumulate melanin (e.g. cells in the brain, and muscles) and in these instances immuno-enhancing or antimicrobial activity defend cells from oxidative stress (Mackintosh, 2001; Riley, 1992).

Of course the Chinese, like all Mulatto people,

are deeply conflicted about what Race they really are...

But regardless of their willingness to accept the truth, the scientific evidence that

modern Mulatto and Albino Mongols are African in origin has been around for many years.

As you can read: according to this study, Mongol people are "PURE" African, without a hint of anything else.

These are the Africans that modern Mulatto and Albino Mongols descend from.

The Chinese, Japanese, and the other Mongol people of Asia are of course not immune to Race isolation and confusion. Blacks, their progenitors, are on the lowest rungs now: thanks to being totally unprepared for the untold millions of Central Asian Dravidian Albinos the Mongols would chase into Europe, thereby completely changing the demographics of Europe. Then being unprepared for the Central Asian Albinos "Clannishness" and capacity for Wanton killing and Murder. The reality of these Albino traits caused the Black man to loose Suzerainty over Europe and the Americas: he just couldn't compete with the Albinos greed and willingness to murder. To civilized Humans, living like that is pointless, so in many cases, they simply accepted death. We often wonder what foul deeds the Albinos did to be chased out of Asia by the Mongols. Perhaps it was simply a matter of the Mongols being more "Perceptive" than Europe's and the Americas Blacks.

Please see the articles about Trump and his people in the "Special Subject Page" "Bits-n-Pieces": these articles clearly show that a large part of the Albino population in the United States has not changed at all - they are still Clannish, hating, and Murderous!

So what a confusing world the Mongol finds himself in: his progenitor, the Black man, is on the ground covered in dust, and his old enemy the Dravidian Albino, is his master. Common sense says that it's the Dravidian Albino he must try to emulate, but at what cost to his "Id". Generically they are known as a people who copy and imitate with precision, but are ill equipped for original thought and invention. This is the result of Chinese Mongols Usurping the Black Xi and Shang militarily, without the intellectual background to move much beyond the Xi and Shang inventions of Gunpowder and Rockets. All that has been left for them is to copy the European Albinos theft of Black invention like the Gun, Windmill, and Steam Engine(Egyptian inventions of the 1200s).

With the Japanese this has manifested itself with their peculiar affection for depictions of themselves: that look "NOTHING" like them, but instead, look like European Albinos!

Japanese Anime

But, whether or not they choose to accept it,

they - like all Mulatto people - derive from a Black person and an Albino.

Remember the Albino lie (and a silly one at that) which said that White people turned White

so they could more easily absorb Sunshine and make Vitamin "D" in the Northern latitudes?

DEBUNKED!

WHAT PALE SKIN (Albinism) DOES PROMOTE IS "SKIN CANCER"!

Well, if you understand that Albinos will always lie about their Albinism,

and look at the people who really are "NATIVE" to the northern latitudes,

you see the truth: THEY ARE ALL DARK SKINNED PEOPLE!

Before moving on, lets be clear exactly what SLC24A5 and SLC45A2 are:

THEY ARE ALBINISM GENES!

Note that the "Other" genes mentioned - are also Albinism genes.

This is the latest lying Albino nonsense study,

which got the Chinese scientists all hopped-up,

to where they decided to add their nonsense two cents worth of denial.

Definition:

SLC24A5 gene - Sodium/potassium/calcium exchanger 5 (NCKX5), also known as solute carrier family 24 member 5 (SLC24A5), is a protein that in humans is encoded by the SLC24A5 gene that has a major influence on natural skin color variation. The NCKX5 protein is a member of the potassium-dependent sodium/calcium exchanger family. Sequence variation in the SLC24A5 gene, particularly a non-synonymous SNP changing the amino acid at position 111 in NCKX5 from alanine to threonine, has been associated with differences in skin pigmentation.

The SLC24A5 gene's derived threonine or Ala111Thr allele (rs1426654) has been shown to be a major factor in the light skin tone of Europeans compared to Africans, and is believed to represent as much as 25–40% of the average skin tone difference between Europeans and West Africans. It has been the subject of recent selection in Europe, and is fixed in European populations.

The derived threonine allele (Ala111Thr; also known as A111T or Thr111) represented 98.7 to 100% of the alleles in European samples, while the ancestral or alanine form was found in 93 to 100% of samples of Sub-Saharan Africans, East Asians and Indigenous Americans.

Above we established that SLC24A5 and SLC45A2 were Albinism genes, but now they have introduced a new term (C11) to confuse people not familiar with genetic science. They tell us that they have decided to call "HAPLOTYPES" with A111T alleles "HAPLOTYPE C11", so now lets define those terms:

(C11) = HAPLOTYPES with A111T alleles

Haplotypes/Haplogroups

A haplotype is a group of genes in an organism that are inherited together from a single parent, and a haplogroup is a group of similar haplotypes that share a common ancestor with a single-nucleotide polymorphism mutation (SNP). More specifically, a haplogroup is a combination of alleles at different chromosomes regions that are closely linked and that tend to be inherited together. As a haplogroup consists of similar haplotypes, it is usually possible to predict a haplogroup from haplotypes. Haplogroups pertain to a single line of descent, usually dating back thousands of years. As such, membership of a haplogroup, by any individual, relies on a relatively small proportion of the genetic material possessed by that individual. Each haplogroup originates from, and remains part of, a preceding single haplogroup (or paragroup). As such, any related group of haplogroups may be precisely modelled as a nested hierarchy, in which each set (haplogroup) is also a subset of a single broader set (as opposed, that is, to biparental models, such as human family trees).

Paragroup

Paragroup is a term used in population genetics to describe lineages within a haplogroup that are not defined by any additional unique markers. In human Y-chromosome DNA haplogroups, paragroups are typically represented by an asterisk (*) placed after the main haplogroup.

The term "paragroup" is a portmanteau of the terms paraphyletic haplogroup indicating that paragroups form paraphyletic subclades. Apart from the mutations that define the parent haplogroup, paragroups may not possess any additional unique markers. Alternatively pargroups may possess unique markers that have not been discovered. If a unique marker is discovered within a paragroup, the specific lineage is given a unique name and is moved out of the paragroup to form an independent subclade.

For example, the paragroup of human Y-DNA Haplogroup DE is DE*. A member of DE* has the marker that defines DE, but not the markers that define DE's only known immediate subclades, haplogroups D and E. Likewise, haplogroup E1b1b1g (also known as E-M293) is an example of a relatively new subclade, discovered within a previously designated paragroup and assigned a new name.

portmanteau

A portmanteau or portmanteau word is a linguistic blend of words, in which parts of multiple words or their phones (sounds) are combined into a new word, as in smog, coined by blending smoke and fog, or motel, from motor and hotel. In linguistics, a portmanteau is defined as a single morph that represents two or more morphemes.

In taxonomy, a group is paraphyletic if it consists of the group's last common ancestor and all descendants of that ancestor excluding a few—typically only one or two—monophyletic subgroups. The group is said to be paraphyletic with respect to the excluded subgroups. The arrangement of the members of a paraphyletic group is called a paraphyly. The term is commonly used in phylogenetics (a subfield of biology) and in linguistics.

The term was coined to apply to well-known taxa like Reptilia (reptiles) which, as commonly named and traditionally defined, is paraphyletic with respect to mammals and birds. Reptilia contains the last common ancestor of reptiles and all descendants of that ancestor—including all extant reptiles as well as the extinct synapsids—except for mammals and birds. Other commonly recognized paraphyletic groups include fish, monkeys and lizards.

Note that Albino people are now so desperate to hide their origins, that they have taken to using "Reptilian" terms to confuse the curious.

The term Basal was formerly used to describe what is now called a "Paragroup".

In general, clade A is more basal than clade B if B is a subgroup of the sister group of A. Within large groups, "basal" may be used more loosely to mean 'closer to the root than the great majority of' and in this context terminology such as "very basal" may arise. A 'core clade' is a clade representing all but the basal clade of lowest rank within a larger clade.

The value of finding Basal Clades or "Paragroups" is best shown with Arabs, American Indians, and the Shang, Jomon and Ainu of China. In these cases, Albinos have killed them, then interbred with the survivors and stolen their identity.

Art of the original settlers of Japan - the Black Jomon - 14,000 – 300 B.C.

Art of the second settlers of Japan - the (formerly) Black Ainu - settled circa 10,000 B.C.

This is what "REAL" American Indians looked like.

It is only with the Socotra Island People: By genetic definition - the Purest Arabs, and probably the closest descendants of the original people of the Arabian Peninsula. They are pure Arab because they are the only people in the world carrying the "Basal form" or "Paragroup" of Y-dna haplogroup "J".

Albinos and their Mulattoes tell us that these^ people are ARAB!

But we already know by common sense and genetics, that THESE people are the true Arabs!

"True" Arabs on the Island of Socotra

Well you say: If those White, and almost White, people above are NOT Arabs...

Then What are they???

TURKS!

So after we have now gotten past the "made-up to confuse terminology".

The lie the Albinos are trying to make, is that the gene mutations

SLC24A5, MC1R, TYR, TYRP1, and OCA2

are SKIN LIGHTNING genes unique to Europeans and East Asians:

Thus Europeans and White East Asians can't be Albinos!

HERE IS THE TRUTH!

It and they - are all African!

Clearly the picture above gives an inaccurate example of the complexion of the Agaw people.

Ann Gibbons Most people associate Africans with dark skin. But different groups of people in Africa have almost every skin color on the planet, from deepest black in the Dinka of South Sudan to beige in the San of South Africa. Now, researchers have discovered a handful of new gene variants responsible for this palette of tones. The study, published online this week in Science, traces the evolution of these genes and how they traveled around the world. While the dark skin of some Pacific Islanders can be traced to Africa, gene variants from Eurasia also seem to have made their way back to Africa. And surprisingly, some of the mutations responsible for lighter skin in Europeans turn out to have an ancient African origin. “This is really a landmark study of skin color diversity,” says geneticist Greg Barsh of the HudsonAlpha Institute for Biotechnology in Huntsville, Alabama. Researchers agree that our early australopithecine ancestors in Africa probably had light skin beneath hairy pelts. “If you shave a chimpanzee, its skin is light,” says evolutionary geneticist Sarah Tishkoff of the University of Pennsylvania, the lead author of the new study. “If you have body hair, you don’t need dark skin to protect you from ultraviolet [UV] radiation.” Until recently, researchers assumed that after human ancestors shed most body hair, sometime before 2 million years ago, they quickly evolved dark skin for protection from skin cancer and other harmful effects of UV radiation. Then, when humans migrated out of Africa and headed to the far north, they evolved lighter skin as an adaptation to limited sunlight. (Pale skin synthesizes more vitamin D when light is scarce.) {This lie is debunked above} Previous research on skin-color genes fit that picture. For example, a “depigmentation gene” called SLC24A5 linked to pale skin swept through European populations in the past 6000 years. But Tishkoff ’s team found that the story of skin color evolution isn’t so black and white. Her team, including African researchers, used a light meter to measure skin reflectance in 2092 people in Ethiopia, Tanzania, and Botswana. They found the darkest skin in the Nilo-Saharan pastoralist populations of eastern Africa, such as the Mursi and Surma, and the lightest skin in the San of southern Africa, as well as many shades in between, as in the Agaw people of Ethiopia. At the same time, they collected blood samples for genetic studies. They sequenced more than 4 million single nucleotide polymorphisms (SNPs)—places where a single letter of the genetic code varies across the genomes of 1570 of these Africans. They found four key areas of the genome where specific SNPs correlate with skin color. The first surprise was that SLC24A5, which swept Europe, is also common in East Africa—found in as many as half the members of some Ethiopian groups. This variant arose 30,000 years ago and was probably brought to eastern Africa by people migrating from the Middle East, Tishkoff says. But though many East Africans have this gene, they don’t have white skin, probably because it is just one of several genes that shape their skin color. The team also found variants of two neighboring genes, HERC2 and OCA2, which are associated with light skin, eyes, and hair in Europeans but arose in Africa; these variants are ancient and common in the light-skinned San people. The team proposes that the variants arose in Africa as early as 1 million years ago and spread later to Europeans and Asians. “Many of the gene variants that cause light skin in Europe have origins in Africa,” Tishkoff says. The most dramatic discovery concerned a gene known as MFSD12. Two mutations that decrease expression of this gene were found in high frequencies in people with the darkest skin. These variants arose about a half-million years ago, suggesting that human ancestors before that time may have had moderately dark skin, rather than the deep black hue created today by these mutations. These same two variants are found in Melanesians, Australian Aborigines, and some Indians. These people may have inherited the variants from ancient migrants from Africa who followed a “southern route” out of East Africa, along the southern coast of India to Melanesia and Australia, Tishkoff says. That idea, however, counters three genetic studies that concluded last year that Australians, Melanesians, and Eurasians all descend from a single migration out of Africa. Alternatively, this great migration may have included people carrying variants for both light and dark skin, but the dark variants later were lost in Eurasians. To understand how the MFSD12 mutations help make darker skin, the researchers reduced expression of the gene in cultured cells, mimicking the action of the variants in dark-skinned people. The cells produced more eumelanin, the pigment responsible for black and brown skin, hair, and eyes. The mutations may also change skin color by blocking yellow pigments: When the researchers knocked out MFSD12 in zebrafish and mice, red and yellow pigments were lost, and the mice’s light brown coats turned gray. “This new mechanism for producing intensely dark pigmentation is really the big story,” says Nina Jablonski, an anthropologist at Pennsylvania State University in State College. The study adds to established research undercutting old notions of race. You can’t use skin color to classify humans, any more than you can use other complex traits like height, Tishkoff says. “There is so much diversity in Africans that there is no such thing as an African race.”

Caution to the gullible and unwary Black person:

even Albinos who seem to be sympathetic and telling the truth,

often hide a lie in there somewhere.

THE SKIN COLOR IS THE SAME!