This is a specific subject page, dealing exclusively with, or primarily with, the subject in the title. Because of need, there are many such pages at RHWW: usually, but not always, linked to primary pages. For those in a hurry, they enable a quick summary of many important subjects. The menu for these pages is here: Click>>>

 

 

Predicting the Skin Color of Children

with One Albino Parent

 

Background:

The first modern humans evolved in Southeast Africa with

Black skin to protect them from the Suns UV radiation.

 

 

 

 

Those first Modern Humans were of the phenotypes that we call today: Negroid, Caucasoid, and Mongoloid.

 

.  

 

 

 

 

 

Over time, their bodies started making mistakes (Mutations) as they replicated themselves with sexual reproduction. The mistakes effecting their skin color, and thus their survivability in the Sun, is collectively called Albinism.

 

Albinism probably started slowly, just one or two partial mutations at first.

 

 

 

 

 

 

 

 

 

 

Then it advanced to Oculocutaneous Albinism type II (OCA2)

 

(OCA2)

 

 

   

 

 

 

 

 

 

Until finally, there was FULL Albinism in Modern Humans.

 

(OCA1)

 

 

 

 

 

 

 

 

 

Albinism effects the following Genes:

 

OCA1A - Oculocutaneous albinism type IA (OCA1A) is caused by homozygous or compound heterozygous mutation in the tyrosinase gene (TYR; 606933) on chromosome 11q14.

OCA1B - Oculocutaneous albinism type IB (OCA1B) is caused by homozygous or compound heterozygous mutation in the tyrosinase gene (TYR; 606933) on chromosome 11q14.

OCA2 - Oculocutaneous albinism type II (OCA2) is caused by mutation in the OCA2 (P) gene (611409).

OCA3 - Oculocutaneous albinism type III (OCA3) is caused by homozygous or compound heterozygous mutation in tyrosinase-related protein-1 (TYRP1; 115501) on chromosome 9p23.

OCA4 - Oculocutaneous albinism type IV (OCA4) is caused by homozygous or compound heterozygous mutation in the MATP gene (SLC45A2; 606202) on chromosome 5p13.

OCA5 - Based on a consensus of the albinism research community (Montoliu et al., 2013), the form of oculocutaneous albinism mapped to chromosome 4q24 is here designated (OCA5).

OCA6 - Oculocutaneous albinism type VI (OCA6) is a type of oculocutaneous albinism (see this term), recently discovered in one Chinese family, characterized by light hair at birth that darkens with age, white skin, transparent irides, photophobia, nystagmus, foveal hypoplasia and reduced visual acuity and that is due to mutations in the SLC24A5 gene (15q21.1).

OCA7 - Based on a consensus of the albinism research community (Montoliu et al., 2013), the form of oculocutaneous albinism caused by mutation in the C10ORF11 gene is here designated (OCA7).

 

 

 

 

 

THIS IS WHAT THE SUN DOES TO PEOPLE WITH ALBINISM!

 

 

   

 

 

So naturally most people with Albinism will only make babies with

normally pigmented people, so that their children will not be Albinos.

 

 

 

 

 

 

 

 

 

 

Except for the Black Dravidians in India - some of their Albinos did indeed mate with each other, and in so doing created the White Race of Humans.

 

Because when an Albino mates with another Albino, the resulting offspring can ONLY be another Albino.

 

Pause History:

 

Remember This?

Healthline Media, Nov 23, 2015 - Albinism is a RARE group of genetic disorders that cause the skin, hair, or eyes

to have little or no color. Albinism is also associated with vision problems.

In their ongoing efforts to distract from their Albinism, Europeans have tried to convince the rest of the world that Albinism is a very RARE condition, too rare for it to account for their large population. But the data provided by media like this publication has forced them to change their tune, just a bit...

 

National Organization for Albinism and Hypopigmentation

Information Bulletin - What is Albinism?

Quote: Albinism is an inherited genetic condition that reduces the amount of melanin pigment formed in the skin, hair and/or eyes. Albinism occurs in all racial and ethnic groups throughout the world. In the U.S., approximately one in 18,000 to 20,000 people has some type of albinism. In other parts of the world, the occurrence can be as high as one in 3,000. Most children with albinism are born to parents who have normal hair and eye color for their ethnic backgrounds.

A common myth is that people with albinism have red eyes. Although lighting conditions can allow the blood vessels at the back of the eye to be seen, which can cause the eyes to look reddish or violet, most people with albinism have blue eyes, and some have hazel or brown eyes. There are different types of albinism and the amount of pigment in the eyes varies; however, vision problems are associated with albinism.

 

 

Making an Albino:

 

Put in it's simplest terms, an Albino can only be produced when a Man with a "Mutated" Albinism gene (though having normal looking Black skin), mates with a Woman with a "Mutated" Albinism gene (though having normal looking Black skin). Their chances of producing an Albino is indicated by the following graphic.

 

 

 

As can be clearly seen, ONE child will be an Albino, ONE child will be Black and having "Normal" genes, but TWO will have Black skin and "Mutated" genes, and thus be Carriers of Albinism.

 

 

However, that is of course a mathematical prediction: meaning that over the course of a sufficient number of sample cases, those ratios would hold. But as seen in the case below, this Albino mother and her Black (carrier) Husband produced THREE Albino children, and only ONE normally pigmented Black child.

 

 

 

Multiple Albino births is common with ONE Albino parent and one Black (carrier) parent,

AND two BLACK (carrier) parents.

So No, Albinism is not Rare!

 

 

 

 

In order to produce Mulattoes (people with protective levels of Melanin),

an Albino must mate with a Black or a Mulatto: for safety, preferably a non-carrier.

 

But to repeat - When an Albino is mating with another Albino,

the resulting offspring can ONLY be another Albino.

 

 

 

 

Resume History:

 

Eventually India's Albinos moved north into Central Asia where they continued to breed with each other. Over time, they had created enough of themselves to qualify as a separate race - the White or Albino race. They all-along mixed with their neighbors, the Black Mongol type people in East Asia. Thereby producing mulattoes among them, until finally, the Mongols too qualified as a separate race.

 

 

   

 

 

At about 1,500 B.C. India's Albinos returned to India as Invaders. About 300 years later, they also invaded Black Europe. These invaders were eventually assimilated into Black Europe and harmony was restored.

However, at about the turn of the modern era, Central Asia's Albinos were chased out of Asia by the Mongol people. Those Albinos were the ancestors of the modern Albinos that we know today: The Germanics, the Slav's, and the Turk's.

 

We know for sure that they were India's Albinos because of the description given by the Roman historian Tacitus.

 

Tacitus: Germany Book 1 [1]

4. For my own part, I agree with those who think that the tribes of Germany are free from all taint of intermarriages with foreign nations, and that they appear as a distinct, unmixed race, like none but themselves. Hence, too, the same physical peculiarities throughout so vast a population. All have fierce blue eyes, red hair, huge frames, fit only for a sudden exertion. They are less able to bear laborious work. Heat and thirst they cannot in the least endure; to cold and hunger their climate and their soil inure them.

 

Eventually Asia was emptied of all it's Indian Albinos: and in turn, those many millions of Albinos were sufficient to overwhelm Europe's native Blacks, and eventually the Albinos forced out all of Europe's native Black people: leaving only mulattoes for the Albinos to mate with. But when mating with a Mulatto, the offspring of the Albino will be moving successively closer to Albino, rather than to fully normal Black.

 

We know anecdotally that a Mulatto will produce progressively Whiter or Blacker offspring, depending on whether he/she mates with Albinos or Blacks. But once total outward Whiteness is achieved (Europeans), does that also mean that the genetic defect that caused Albinism is now total, or is there still some "Normal" (Black) genes in that White person?

 

 

 

 

 

The case of Sandra Laing - (a genetic "Return to Normalcy")?

 

Sandra Laing was born in 1955 to Sannie and Abraham Laing, Afrikaners in Piet Retief, a small conservative town in South Africa during the apartheid era, when laws governed officially established social castes of racial classification. The girl had darker skin than others in her family, which seemed to become more obvious as she grew older. Her parents, grandparents, and great-grandparents were all white, but Sandra displayed the physiognomy of African ancestors of earlier generations, perhaps from the 18th century or more recent. Her family treated her as white, the same as their sons Adriaan and Leon, and together they all attended the Dutch Reformed Church.

 

 

 

 

When Sandra was 10 years old and at an all-white boarding school, the school authorities expelled her because of complaints from the parents of other students, based on her appearance: primarily her skin colour and the texture of her hair. They believed she was "coloured", a term for mixed-race people. She was expelled and escorted home by two police officers.

Sandra's parents fought several legal battles to have her classified as white, based on her documented ancestry through them. Her father underwent a blood-typing test for paternity in the 1960s, as DNA tests were not yet available. The results were compatible with his being her biological father, though such tests are extremely imprecise due to the small number of blood types that most people have.

After the publicity, Sandra found herself shunned by the white community, although she was re-classified as white again in 1966 when the law was changed to allow a person to be classified as white if both parents are classified as white. She attended a coloured boarding school away from her family and became immersed in the non-white world. Her only friends were the children of black employees. At the age of 16, Laing eloped to Swaziland with Petrus Zwane, a black South African who spoke Zulu.

She was jailed for three months for illegal border-crossing. Her father threatened to kill her for the marriage and broke off contact with her. They never met again.

 

 

 

 

Although she and her husband had two children, who were classified as "coloured", she was threatened with losing them unless she also was classified as "coloured", as a white parent could not raise coloured children. At the age of 26, she arranged for the change in race classification officially, although her father had refused permission earlier. Except for secret trips to see her mother when her father was out of the house, Laing was estranged from her family and struggled to survive economically. When her parents moved away from Piet Retief, the clandestine visits were no longer possible. Laing lost contact with her family completely.

 

 

 

 

Laing and her husband separated due to the pressures they were under, and she put their children into government care for a period. Years later she married again, to Johannes Motloung, a Sotho-speaking man. They had three children together and she was able to reclaim her first two; all are now grown and with families of their own. Trying to reconcile with her family in the 1980s, Laing learned that her father had died and her mother Sannie refused to see her.

 

 

 

 

In 2000 the Johannesburg Times tracked down Laing to learn about her years since the end of apartheid. The newspaper helped her find her mother, and they were able to reconcile. Sannie was then in a nursing home. Sannie and she shared time together before her mother's death in 2001.

 

 

 

 

 

Naturally these almost limitless possible combinations of normally pigmented humans, partially pigmented humans, and Albinos, produces a dizzying array of variations in human skin color.

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

Luckily modern science allows us to predict what the skin color

of many combinations of Black/Mulatto/Albino will be.

 

 

One of the simplest is the prediction table of

Charles Benedict Davenport (June 1, 1866 – February 18, 1944).

 

 

 

As we can clearly see from Davenports table, the MAJORITY of Europeans are supposed to be BROWN skinned Mulattoes!

And according to Benjamin Franklin - THEY WERE!

 

 

 

Black = Black

Tawny = a shade of brown tinged with yellow

 

So what is Swarthy?

Wiki - Swarthy = old High German also had two words for black: swartz for dull black and blach for a luminous black. These are paralleled in Middle English by the terms swart for dull black and blaek for luminous black. Swart still survives as the word swarthy, while blaek became the modern English black.

Webster's - Swarthy = of a dark color, complexion, or cast.

Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 - Swarthy = dark-hued or dark-complexioned.

 

 

 

 

 

 

 

SO WHAT HAPPENED?

 

As Europe was EMPTIED of Blacks after the many Race Wars of the late Medieval period: whether by MURDER, or EXPULSION to the Americas as SLAVES or INDENTURED SERVANTS. The only mating choice for Europe's Mulattoes was with FULL Albinos. The result of that mating pattern was the gradual WHITENING of Europe's populations.

 

 

Today humans freely transform between Black and Albino derived White.

 

 

 

 

 

 

 

This is the Croatian skin color predictor.

 

 

 

 

 

 

 

Click for Realhistoryww Home Page